Exploiting Cooperative Pathogen Behavior for Enhanced Antibiotic Potency: A Trojan Horse Approach.
Mutlu A, Vanderpool E, Rumbaugh KP, Diggle SP, Griffin AS.

High prevalence of lipopolysaccharide mutants and R2-pyocin susceptible variants in Pseudomonas aeruginosa populations sourced from cystic fibrosis lung infections.
Mei M, Pheng P, Kurzeja-Edwards D, Diggle SP.

The role of hypermutation and collateral sensitivity in antimicrobial resistance diversity of Pseudomonas aeruginosa populations in cystic fibrosis lung infection.
Vanderwoude J, Azimi S, Read T, Diggle SP.

Frequency of quorum-sensing mutations in Pseudomonas aeruginosa strains isolated from different environments.
O'Connor K, Zhao CY, Mei M, Diggle SP.

O-Specific Antigen-Dependent Surface Hydrophobicity Mediates Aggregate Assembly Type in Pseudomonas aeruginosa.
Azimi S, Thomas J, Cleland SE, Curtis JE, Goldberg JB, Diggle SP.

Heterogenous Susceptibility to R-Pyocins in Populations of Pseudomonas aeruginosa Sourced from Cystic Fibrosis Lungs.
Mei M, Thomas J, Diggle SP.

The evolution of virulence in Pseudomonas aeruginosa during chronic wound infection.
Vanderwoude J, Fleming D, Azimi S, Trivedi U, Rumbaugh KP, Diggle SP.

Allelic polymorphism shapes community function in evolving Pseudomonas aeruginosa populations.
Azimi S, Roberts AEL, Peng S, Weitz JS, McNally A, Brown SP, Diggle SP.

Conflict of interest and signal interference lead to the breakdown of honest signaling.
Popat R, Pollitt EJ, Harrison F, Naghra H, Hong KW, Chan KG, Griffin AS, Williams P, Brown SP, West SA, Diggle SP.

Recombination is a key driver of genomic and phenotypic diversity in a Pseudomonas aeruginosa population during cystic fibrosis infection.
Darch SE, McNally A, Harrison F, Corander J, Barr HL, Paszkiewicz K, Holden S, Fogarty A, Crusz SA, Diggle SP.